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Think your Stressed?

March 28, 2009

Stressed Molecular Membranes
The uptake and distribu tion mechanism of anesthetics is
known (Eger, 1998). The previously accepted theory ascribed to the action of
anesthetics, namely “molecular membrane stress” applied to the bi-lipid layer
of cell membranes (Ueda, 2001), could conceivably be applied to explain some
of the actions of essential oils at the cellular membrane level. A predominantly
accepted theory for the contiguous mechanisms of consciousness and
anesthesia results from the weak quantum interactions of van der Waals and
London forces acting in hydrophobic pockets of proteins that link synaptically
integrated dendrites of brains cells. Many essential oils and many anesthetic
molecules are aliphatic hydrocarbon chains (Hameroff, 2006). Some essential
oils are rich in molecules that are steroidal in structure.
The natural plasticity of the bi-lipid layers of cell walls and organelles
of various body tissues is due in part to the orientation of the hydrophilic
and hydrophobic lipid layers. The cell membranes have abundant embedded
essential sugar-coated glycoprotein receptors for cell-to-cell immunity
communication and gated ligand ionic channels thought to be acted upon by
the various volatile chemicals of the anesthetic gas agents.

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